alpha-keto-gamma-methylthiobutyric acid

Quinovic acid 3-O-alpha-L-rhamnopyranoside shows significant inhibitory activity against snake venom phosphodiesterase-I.

Oleanolic acid 3-O-beta-D-glucosyl-(1->3)-alpha-L-rhamnosyl(1->2)-alpha-L-arabinoside is a natural product

3-Acetyl-11-keto-ursolic acid (Compound 18), a triterpenoid derived from Eriobotrya japonica leaves, exhibits 11B-hydroxysteroid dehydrogenase 1 (11b-HSD1) inhibitory activity and holds potential for anti-diabetes research [1].

alpha-Epoxydihydroartemisinic acid is a natural product for research related to life sciences. The catalog number is TN3384 and the CAS number is 380487-65-0.

alpha-Boswellic acid is a pentacyclic triterpenoid obtained from Boswellia serrata with anticonvulsant and anticancer properties. In animals, it is a preventive and minimizing Alzheimer's disease marker and can be used in Alzheimer's disease research.

7-keto-deoxycholic acid is converted from Lactobacillus and Bifidobacterium with specific condition and from cholic acid in vitro.

Danshensu has antioxidant activity, can enhance HO-1 expression to suppress 6-OHDA-induced oxidative damage via PI3K/Akt/Nrf2 signaling pathways.

3-O-methylellagic acid 4'-O-alpha-L-rhamnopyranoside is a natural product for research related to life sciences. The catalog number is TN6494 and the CAS number is 51768-39-9.

11-Keto-beta-boswellic acid, a novel Nrf2 activator, and a selective 5-lipoxygenase (5-LOX) inhibitor; it exerts dose dependent cardioprotective effect manifested by dose-dependent reduction in serum lactate dehydrogenase

gamma-Linolenic acid (gamolenic acid), is an unsaturated fatty acid synthesized from linoleic acid (LA) by the enzyme delta-6-desaturase. Research on hepatocellular cell lines has shown γ-Linolenic Acid to have induced reactive oxygen species generation including lipid peroxidation, cell growth inhibition, and heme oxygenase-1 production for antioxidant protection against oxidative stress. Further experiments have noted that γ-Linolenic acid inhibits inflammatory responses through inactivation of NFκB and activator protein-1 by suppressed oxidative stress.