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(1)
By Target- Autophagy
- Ferroptosis(1)
- HMG-CoA Reductase(1)
- Others(3)
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Biochemicals
- Autophagy
- Cell Cycle/Checkpoint
- Cytoskeletal Signaling
- Chromatin/Epigenetic
- Endocrinology/Hormones
- Hedgehog/Smoothened
- Angiogenesis
- DNA Damage/DNA Repair
- Apoptosis
- NF-κB
- Neuroscience
- MAPK
- Microbiology/Virology
- GPCR/G Protein
- TGF-beta/Smad
- Immunology/Inflammation
- Proteases/Proteasome
- Stem Cells
- Ubiquitination
- Oxidation-Reduction
- Membrane transporter/Ion channel
- PI3K/Akt/mTOR signaling
- Metabolism
- Wnt/beta-catenin
- JAK/STAT signaling
- Tyrosine Kinase/Adaptors
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- Casein Kinase
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- Dehydrogenase
- DHFR
- Drug Metabolite
- Endogenous Metabolite
- Epoxide Hydrolase
- FAAH
- FABP
- Factor Xa
- Fatty Acid Synthase
- FXR
- Glucokinase
- Glucosidase
- Glutathione Peroxidase
- Glycosidase
- GSNOR
- hCE
- Hexokinase
- HIF/HIF Prolyl-Hydroxylase
- HMG-CoA Reductase
- HSP
- Hydrogenase
- Hydroxylase
- IDO
- Indoleamine 2,3-Dioxygenase (IDO)
- Isocitrate Dehydrogenase (IDH)
- LDL
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- Lipoxygenase
- Liver X Receptor
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Results for "
pravastatin
" in TargetMol Product Catalog- Inhibitors & Agonists4
- Natural Products1
Pravastatin
Cited
T2240581093-37-0
Pravastatin is a lipoprotein-lowering drug via reversibly inhibiting hydroxymethylglutaryl-CoA (HMG-CoA) reductase and the synthesis of very-low-density lipoproteins.
- Inquiry Price
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QTY
CitedPravastatin sodium Cited
CS-514 Sodium,CS-514 (sodium)
T067281131-70-6
Pravastatin sodium (CS-514 (sodium)), an HMG-CoA reductase inhibitor, inhibits sterol synthesis with IC50 of 5.6 μM.
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Cited3α-Hydroxy pravastatin sodium
T4091381093-43-8
3α-Hydroxy pravastatin sodium, the primary metabolite of Pravastatin, is a potent competitive inhibitor of HMG-CoA reductase.
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Pravastatin lactone
T3764285956-22-5
Pravastatin lactone is a metabolite of pravastatin , a hydroxymethylglutaryl-coenzyme A (HMG-CoA) reductase inhibitor that is a ring-hydroxylated metabolite of mevastatin . Pravastatin lactone is formed when pravastatin undergoes acid-catalyzed non-enzymatic lactonization in the stomach following oral administration.
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